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Not everyone suspected to have PE should have a V/Q scan. The attending clinicians
will decide if this is the appropriate course of action. There are a lot more mundane
conditions which could mimic this serious condition, including such things as muscle
or ligament sprain, pressure pain from the fetal limbs in late pregnancy (under the
ribs) and some self-
MRI is also increasingly used as a diagnostic tool for suspected PE.
A chest X-
The mainstay of treatment for PE in pregnancy is the anticoagulant heparin. In pulmonary embolism, this will be given in the form of continuous infusion at a much higher dose than that used in uncomplicated deep vein thrombosis. Special tests will be carried out regularly to ensure that the right effective dose is being given. This may continue for a few days before switching to heparin/LMWH injections. Treatment will continue for the remainder of the pregnancy and for at least six weeks after delivery but this could be longer, even several months. Assessment of risk is made to determine this.
In the majority of cases, the treatment described above is successful. In some very
serious cases, this may be insufficient and very specialized surgical intervention
may be necessary as a life-
Heparin does not cross the placenta and therefore, as far as the baby is concerned, it is perfectly safe. Regular tests will be carried out to ensure that an optimal dose is being given to the mother.
Too much heparin can cause bleeding tendencies and, if tests show evidence of this, the dose will be adjusted downwards.
Another problem associated with prolonged heparin use has been the risk of osteoporosis. In rare instances, this has been known to lead to bone fractures.
However, the benefits of treatment far outweigh the potential risk.
LMWH are also a risk factor for osteoporosis but to a much lower degree. There is a number of LMWH preparations. Common brands include Fragmin® (Dalteparin), Clexane® (Enoxaparin) and Innohep® (Tinzaparin).
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